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A transcriptomic computational analysis of mastic oil-treated Lewis lung carcinomas reveals molecular mechanisms targeting tumor cell growth and survival

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dc.contributor.author Moulos, P en
dc.contributor.author Papadodima, O en
dc.contributor.author Chatziioannou, A en
dc.contributor.author Loutrari, H en
dc.contributor.author Roussos, C en
dc.contributor.author Kolisis, F en
dc.date.accessioned 2014-03-01T01:57:42Z
dc.date.available 2014-03-01T01:57:42Z
dc.date.issued 2009 en
dc.identifier.uri https://dspace.lib.ntua.gr/xmlui/handle/123456789/28476
dc.subject Biological Process en
dc.subject Cell Cycle en
dc.subject Cell Line en
dc.subject Cell Proliferation en
dc.subject Computer Analysis en
dc.subject Expression Profile en
dc.subject Gene Expression en
dc.subject Inflammatory Response en
dc.subject k-means clustering en
dc.subject Mechanism of Action en
dc.subject Molecular Mechanics en
dc.subject Promoter Analysis en
dc.subject Tumor Cells en
dc.subject Tumor Growth en
dc.subject Lewis Lung Carcinoma en
dc.subject Time Dependent en
dc.title A transcriptomic computational analysis of mastic oil-treated Lewis lung carcinomas reveals molecular mechanisms targeting tumor cell growth and survival en
heal.type journalArticle en
heal.identifier.primary 10.1186/1755-8794-2-68 en
heal.identifier.secondary http://dx.doi.org/10.1186/1755-8794-2-68 en
heal.publicationDate 2009 en
heal.abstract BACKGROUND: Mastic oil from Pistacia lentiscus variation chia, a blend of bioactive terpenes with recognized medicinal properties, has been recently shown to exert anti-tumor growth activity through inhibition of cancer cell proliferation, survival, angiogenesis and inflammatory response. However, no studies have addressed its mechanisms of action at genome-wide gene expression level. METHODS: To investigate molecular mechanisms triggered by mastic oil, en
heal.journalName BMC Medical Genomics en
dc.identifier.doi 10.1186/1755-8794-2-68 en


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