Neuroendocrine manifestations in Sjogren's syndrome

Johnson, EO; Skopouli, FN; Moutsopoulos, HM

dc.contributor.author	Johnson, EO	en
dc.contributor.author	Skopouli, FN	en
dc.contributor.author	Moutsopoulos, HM	en
dc.date.accessioned	2014-03-01T11:45:45Z
dc.date.available	2014-03-01T11:45:45Z
dc.date.issued	2000	en
dc.identifier.issn	0889-857X	en
dc.identifier.uri	https://dspace.lib.ntua.gr/xmlui/handle/123456789/37603
dc.subject.classification	Rheumatology	en
dc.subject.other	PITUITARY-ADRENAL AXIS	en
dc.subject.other	CORTICOTROPIN-RELEASING HORMONE	en
dc.subject.other	SYSTEMIC LUPUS-ERYTHEMATOSUS	en
dc.subject.other	RHEUMATOID-ARTHRITIS	en
dc.subject.other	BEHAVIORAL HOMEOSTASIS	en
dc.subject.other	AUTOIMMUNE-DISEASES	en
dc.subject.other	THYROID-DISEASE	en
dc.subject.other	LEWIS RATS	en
dc.subject.other	PROLACTIN	en
dc.subject.other	STRESS	en
dc.title	Neuroendocrine manifestations in Sjogren's syndrome	en
heal.type	other	en
heal.language	English	en
heal.publicationDate	2000	en
heal.abstract	Sjogren's Syndrome (SS) is a chronic autoimmune disorder characterized by diminished lacrimal and salivary gland secretion. Female SS patients indicated a central deficiency in three neuroendocrine axes: adrenal, gonadal, and thyroid. At present, it is not clear if any one system plays a primary role in the expresson of the disease. Rather, it is likely that the net effect involves the synergistic and antagonistic effects of multiple hormones. When taken together, adrenal and gonadal steroid hormone deficiency, plus elevated prolactin levels probably greatly facilitate cellular immunity in SS patients.	en
heal.publisher	W B SAUNDERS CO	en
heal.journalName	RHEUMATIC DISEASE CLINICS OF NORTH AMERICA	en
dc.identifier.isi	ISI:000165078500014	en
dc.identifier.volume	26	en
dc.identifier.issue	4	en
dc.identifier.spage	927	en
dc.identifier.epage	+	en